Antitumor efficacy of a bispecific antibody that targets HER2 and activates T cells.

نویسندگان

  • Teemu T Junttila
  • Ji Li
  • Jennifer Johnston
  • Maria Hristopoulos
  • Robyn Clark
  • Diego Ellerman
  • Bu-Er Wang
  • Yijin Li
  • Mary Mathieu
  • Guangmin Li
  • Judy Young
  • Elizabeth Luis
  • Gail Lewis Phillips
  • Eric Stefanich
  • Christoph Spiess
  • Andrew Polson
  • Bryan Irving
  • Justin M Scheer
  • Melissa R Junttila
  • Mark S Dennis
  • Robert Kelley
  • Klara Totpal
  • Allen Ebens
چکیده

Clinical results from the latest strategies for T-cell activation in cancer have fired interest in combination immunotherapies that can fully engage T-cell immunity. In this study, we describe a trastuzumab-based bispecific antibody, HER2-TDB, which targets HER2 and conditionally activates T cells. HER2-TDB specifically killed HER2-expressing cancer cells at low picomolar concentrations. Because of its unique mechanism of action, which is independent of HER2 signaling or chemotherapeutic sensitivity, HER2-TDB eliminated cells refractory to currently approved HER2 therapies. HER2-TDB exhibited potent antitumor activity in four preclinical model systems, including MMTV-huHER2 and huCD3 transgenic mice. PD-L1 expression in tumors limited HER2-TDB activity, but this resistance could be reversed by anti-PD-L1 treatment. Thus, combining HER2-TDB with anti-PD-L1 yielded a combination immunotherapy that enhanced tumor growth inhibition, increasing the rates and durability of therapeutic response.

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عنوان ژورنال:
  • Cancer research

دوره 74 19  شماره 

صفحات  -

تاریخ انتشار 2014